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Biomedical Research
Funded in 2001 1) Jeanine Wiener-Kronish, MD, University of California San Francisco, San Francisco, California, Principal Investigator and Kendra Rumbaugh, PhD, Post Doctoral Fellow |
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"Analysis of dendrimeric Carbohydrates as Potential Therapies
for Blocking P. aeruginosa Colonization of the Lungs" |
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This study seeks to determine the genomic analysis of Pseudomonas aeruginosa pathogensis, |
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which is the number one cause of death in CF patients. They plan to examine the host's |
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immune system to determine why CF lungs do not readily clear the bacteria from their lungs, |
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as non-CF lungs do. |
Studies suggest that both the infection site as well as the duration of |
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infection influence the virulence of the mentioned bacteria by altering the production of extracellular virulence factors. |
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Amount
Funded: $35,000.00 |
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2) Terry
Machen, PhD, Christian Schwarzer, PhD, Post Doctoral Fellow |
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"Proton
Channels and Acid Secretions in CF Airways" |
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The purpose of this study was to determine the cause of tissue damage resulting from the |
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inflammatory response in CF airways. The cell hypothesized to be the key player proved to |
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be insignificant, thus more differentiated cells are now being tested in hopes of showing some |
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significance. Additionally, Vitamin C has been found to play a regulatory role in transport |
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across the human airways. The potential benefits of specific products of the airway cells are |
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still unknown, but could possibly help in host defense and bacteria killing. Further research |
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is required to determine that. Amount Funded: $35,000.00 3) George Caughey, MD, University of California San Francisco, San Francisco, California, Principal Investigator and Zhenyue Tong, MD, PhD, Post Doctoral Fellow |
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"Regulation of
Airway Epithelial Ion Flux by Membrance-Anchored Proteases" |
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The goal of this study is to help focus development of pharmaceutical approaches to |
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normalizing salt and water flux in CF airways. The focus is on prostasin, an ENaC-activating |
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protease in humans, and determining whether it aids in sodium transport in human airways. |
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Excessive transport of sodium ions across cells lining the bronchi of CF patients is believed |
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to be a major cause of airway infection. Inhibition of proteases would be helpful in treating CF. Amount Funded: $35,000.00 |
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4) Marybeth Howard, PhD, University of California San Francisco, San Francisco, California, Principal Investigator and Jeremie Roux, Post Doctoral Fellow |
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"The CF
Transmembrane Conductance Regulator Protein and Its Function in Maintenance
of Ion Homesostasis" |
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This study was conducted to determine if the extra-cellular sodium chloride effects the |
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overall maturation of wild type CFTR, which in turn will further explain CFTR's role in ion |
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homeostasis. Results show that there is inefficient maturation of CFTR due to misfolding. |
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Other experiments also suggest that wild type CFTR does not misfold. Further research |
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will be done to define and understand the efficiency and kinetics of CFTR maturation |
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in response to short periods of salt stresses. Further research has also shown that |
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changes in tonicity do not have an effect on the maturation/degradation of the CFTR protein |
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in polarized MDCK cells. Amount Funded: $35,000.00 |
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